![]() ![]() The glob or “dragon” is “chased” with the lighter underneath while the vapor is sucked through a straw or pipe. ![]() The heroin liquefies into a reddish-brown glob, which moves around on the foil and emits a white vapor. A small quantity of powder is placed on aluminum foil, which is then heated underneath with a lighter or matches. "The term “chasing the dragon” appears to have originated in China in the 1920s and became a popular mechanism of heroin administration in the 1950s in Hong Kong because the drug was cheap but impure. The following passage is from the original description in the American Journal of Roentgenology by Ciaran F. May show abnormally elevated intracerebral lactate in the affected white matter as well as decreased levels of N-acetylaspartic acid (NAA) in the white matter, gray matter, and cerebellum. Typically regions demonstrate high DWI signal, which only sometimes represents true diffusion restriction. sparing of adjacent grey matter structures.symmetrical butterfly wing pattern involvement of the cerebellar white matter.Discover live and local AM and FM radio stations near you and from cities around the world. Stream unlimited music, thousands of radio stations and podcasts all in one app. subcortical U-fibers are also usually spared Get more of the radio, music and podcasts you love on the iHeartRadio app.symmetrical involvement of the posterior limb of the internal capsules extending inferiorly into the pontine corticospinal tracts, and superiorly into the perirolandic subcortical white matter.The characteristic distribution includes: These are similar to other toxic leukoencephalopathies with widespread white matter hyperintensity involving both supra and infratentorial compartments. There is symmetric spongiform degeneration, specifically in the cerebral and cerebellar white matter as well as the corticospinal and solitary tracts. There may be a latent period with a subclinical evolution of white matter degeneration. Progression of the disease continues even after cessation of the toxin for up to 6 months. Only a minority of patients progress through all three. spasms, hypotonic paresis, and ultimately death.cerebellar signs and motor restlessness. ![]()
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